The RAS/Raf1/MEK/ERK signaling pathway facilitates VSV-mediated oncolysis: Implication for the defective interferon response in cancer cells

Josh A. Noser; Amber A. Mael; Ryuta Sakuma; Seiga Ohmine; Paola Marcato; Patrick W.K. Lee; Yasuhiro Ikeda

doi:10.1038/sj.mt.6300193

The RAS/Raf1/MEK/ERK signaling pathway facilitates VSV-mediated oncolysis: Implication for the defective interferon response in cancer cells

Josh A. Noser, Amber A. Mael, Ryuta Sakuma, Seiga Ohmine, Paola Marcato, Patrick W.K. Lee, Yasuhiro Ikeda

科研成果: 期刊稿件 › 文章 › 同行评审

源语言	英语
页（从-至）	1531-1536
页数	6
期刊	Molecular Therapy
卷	15
期	8
DOI	https://doi.org/10.1038/sj.mt.6300193
出版状态	已出版 - 8月 2007

!!!ASJC Scopus Subject Areas

分子药学
分子生物学
遗传学
药理学
药物发现

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Noser, J. A., Mael, A. A., Sakuma, R., Ohmine, S., Marcato, P., Lee, P. W. K., & Ikeda, Y. (2007). The RAS/Raf1/MEK/ERK signaling pathway facilitates VSV-mediated oncolysis: Implication for the defective interferon response in cancer cells. Molecular Therapy, 15(8), 1531-1536. https://doi.org/10.1038/sj.mt.6300193

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title = "The RAS/Raf1/MEK/ERK signaling pathway facilitates VSV-mediated oncolysis: Implication for the defective interferon response in cancer cells",

author = "Noser, {Josh A.} and Mael, {Amber A.} and Ryuta Sakuma and Seiga Ohmine and Paola Marcato and Lee, {Patrick W.K.} and Yasuhiro Ikeda",

note = "Funding Information: We express our appreciation and gratitude to Glen N. Barber (University of Miami School of Medicine) for providing VSV-expressing GFP and to Channing J. Der (University of North Carolina at Chapel Hill) for providing the mutant RAS constructs. We thank Stephen J. Russell and Roberto Cattaneo (Mayo Clinic) for helpful discussions. Funding for this work was provided by the Mayo Foundation for Research and Education.",

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AU - Sakuma, Ryuta

AU - Ohmine, Seiga

AU - Marcato, Paola

AU - Lee, Patrick W.K.

AU - Ikeda, Yasuhiro

N1 - Funding Information: We express our appreciation and gratitude to Glen N. Barber (University of Miami School of Medicine) for providing VSV-expressing GFP and to Channing J. Der (University of North Carolina at Chapel Hill) for providing the mutant RAS constructs. We thank Stephen J. Russell and Roberto Cattaneo (Mayo Clinic) for helpful discussions. Funding for this work was provided by the Mayo Foundation for Research and Education.

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The RAS/Raf1/MEK/ERK signaling pathway facilitates VSV-mediated oncolysis: Implication for the defective interferon response in cancer cells

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