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p53 binding to the p21 promoter is dependent on the nature of DNA damage
Richard Hill, Ewelina Bodzak, Michael D. Blough,
Patrick W.K. Lee
Biochemistry, Genetics and Molecular Biology
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探究 'p53 binding to the p21 promoter is dependent on the nature of DNA damage' 的科研主题。它们共同构成独一无二的指纹。
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Keyphrases
DNA Damage
100%
P21 Promoter
100%
Ionizing Radiation
71%
Damaged Cells
42%
Apoptosis
28%
P21 Protein
28%
Protein Level
14%
Signaling Pathway
14%
Chromatin Immunoprecipitation (ChIP)
14%
Post-translational Modification
14%
Tumor Suppressor
14%
Radiation Therapy
14%
Cell Fate
14%
Stress-induced
14%
DNA Repair
14%
Transcriptional Regulation
14%
Cell Cycle Arrest
14%
Anti-apoptotic
14%
Senescence
14%
TATA Box
14%
Histone Acetylation
14%
Transcriptional Targets
14%
Ultraviolet Treatment
14%
Cell Cycle Control
14%
Cell Cycle Profile
14%
Apoptotic Response
14%
Bias against
14%
Cellular Stress
14%
Biochemistry, Genetics and Molecular Biology
Promoter Region
100%
P53
100%
DNA Damage
100%
P21
100%
Tumor Suppressor Protein
9%
Posttranslational Modification
9%
Transcription
9%
Signal Transduction
9%
DNA Damage Response
9%
DNA Repair
9%
Cell Cycle Arrest
9%
Chromatin Immunoprecipitation
9%
TATA Box
9%
Histone Acetylation
9%
Cell Cycle
9%
Cell Fate
9%
Ultraviolet Radiation
9%
Cellular Stress Response
9%
Cell Cycle Regulation
9%
Neuroscience
P53
100%
Promoter Region
100%
DNA Damage
100%
Protein P21
18%
Radiation Therapy
9%
Cell Signaling
9%
Cell Fate
9%
Posttranslational Modification
9%
Cell Cycle
9%
Cell Cycle Arrest
9%
DNA Damage Response
9%
Cellular Stress Response
9%
Immunoprecipitation
9%
TATA Box
9%
Histone Acetylation
9%
Cell Cycle Regulation
9%
DNA Repair
9%
Immunology and Microbiology
Promoter Region
100%
P53
100%
DNA Damage
100%
Chromatin Immunoprecipitation
9%
Cell Fate
9%
Protein Processing
9%
DNA Repair
9%
DNA Damage Response
9%
Cell Cycle Arrest
9%
Cell Cycle
9%
Signal Transduction
9%
Cellular Stress Response
9%
Ultraviolet Radiation
9%
Cell Cycle Regulation
9%
TATA Box
9%
Histone Acetylation
9%