Molecular mechanisms by which iNOS uncoupling can induce cardiovascular dysfunction during sepsis: Role of posttranslational modifications (PTMs)

Nivin Sharawy; Christian Lehmann

doi:10.1016/j.lfs.2020.117821

Molecular mechanisms by which iNOS uncoupling can induce cardiovascular dysfunction during sepsis: Role of posttranslational modifications (PTMs)

科研成果: 期刊稿件 › 文献综述 › 同行评审

源语言	英语
文章编号	117821
期刊	Life Sciences
卷	255
DOI	https://doi.org/10.1016/j.lfs.2020.117821
出版状态	已出版 - 8月 15 2020
已对外发布	是

!!!ASJC Scopus Subject Areas

一般生物化学、遗传与分子生物学
药理学、毒理学和药学（全部）

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10.1016/j.lfs.2020.117821

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title = "Molecular mechanisms by which iNOS uncoupling can induce cardiovascular dysfunction during sepsis: Role of posttranslational modifications (PTMs)",

author = "Nivin Sharawy and Christian Lehmann",

note = "Funding Information: This work was supported by Faculty of Medicine, Cairo University . Publisher Copyright: {\textcopyright} 2020 Elsevier Inc.",

year = "2020",

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day = "15",

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volume = "255",

journal = "Life Sciences",

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AU - Sharawy, Nivin

AU - Lehmann, Christian

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SN - 0024-3205

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JO - Life Sciences

JF - Life Sciences

M1 - 117821

ER -

Molecular mechanisms by which iNOS uncoupling can induce cardiovascular dysfunction during sepsis: Role of posttranslational modifications (PTMs)

!!!ASJC Scopus Subject Areas

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